The cucumbers grown in normal solar greenhouses should be straight, thin cylindrical and slightly apex. In the later stages of cucurbit senescence, abnormal melons are produced and belong to the normal fertility process. When the melons and melons occur in large numbers at the beginning and at the end of melon stage, abnormal fertility occurs. Here's a breakdown of the causes of cucumber deformity introduced to everyone.
The important feature of the curved melon is that the melon strip is seriously bent. The reason for such malformed melons is mainly stems and leaves are too dense, especially when the row spacing is narrow. Sometimes the fertilizer is insufficient, drought and water shortage cause the growth of the plant to be weak, and malnutrition also produces crooked melons; mechanical reasons such as tendril winding and stem vine blocking are also likely to cause crooked melons.
The main characteristic of the bellies is that the head of the melon strip appears to be thick and shaped like a big belly. Such melons are caused by incomplete pollination and fertilization. If you do not experience melancholy melons, the result is that plants are weak and malnourished, especially potassium deficiency. Sometimes in the process of melon growth before and after the lack of water, in the middle of no water can also form a big tummy melon.
The tip of the sharp-edged guagua stripe becomes significantly thinner. Such malformed melons are mostly in the condition of parthenocarpy, continuous high temperature and drought in solar greenhouse, weak plant growth, malnutrition, resulting in melon strips from the middle to the top, inflated elongation is not good, melon strip length is significantly shorter, This formed a sharp-edged melon.
The middle of the thin-waist guagua strips is as thin as a bee and waist, and the cut melon strips show cracked flesh and hollows in the heart. Such deformed melons are generally susceptible to high temperature, drought, and weakening plants. It has also been reported that thin waisted melons may also occur due to potassium deficiency or fertility fluctuations.
Bitter melon is affected by the variety, and the bitter taste in different varieties is different. There is no absolute no bitter taste. However, when the bitterness is increased until it can be clearly paid, it is a melon. It is generally believed that such melons are prone to occur when there are too many nitrogen fertilizers, insufficient water, low temperature, insufficient light, insufficient fertilizer, and weak plantlets at the late growth stage. If a large amount of bitter gourd is produced during the fruiting period, the planting density may be too high and the night temperature may be too high. (Yongqing County Bureau of Agriculture 065600)
Vitamins:
Vitamin A: Retinol. Carotene compounds responsible for transmitting light sensation in the retina of the eye. Deficiency leads to night blindness.
Beta carotene: An antioxidant which protects cells against oxidation damage that can lead to cancer. Beta carotene is converted, as needed, to vitamin A. Food sources of beta carotene include vegetables such as carrots, sweet potatoes, spinach and other leafy green vegetables; and fruit such as cantaloupes and apricots. Excessive carotene in the diet can temporarily yellow the skin, a condition called carotenemia, commonly seen in infants fed largely mushed carrots.
Vitamin B1: Thiamin, acts as a coenzyme in body metabolism. Deficiency leads to beriberi, a disease of the heart and nervous system.
Vitamin B2: Riboflavin, essential for the reactions of coenzymes. Deficiency causes inflammation of the lining of the mouth and skin.
Vitamin B3: Niacin, an essential part of coenzymes of body metabolism. Deficiency causes inflammation of the skin, vagina, rectum and mouth, as well as mental slowing.
Vitamin B6: Pyridoxine, a cofactor for enzymes. Deficiency leads to inflammation of the skin and mouth, nausea, vomiting, dizziness , weakness and anemia.
Folate (folic acid): Folic acid is an important factor in nucleic acid synthesis (the genetic material). Folate deficiency leads to megaloblastic anemia.
Vitamin B12: An essential factor in nucleic acid synthesis (the genetic material of all cells). Deficiency leads to megaloblastic anemia, as can be seen in pernicious anemia.
Vitamin C: Ascorbic acid, important in the synthesis of collagen, the framework protein for tissues of the body. Deficiency leads to scurvy, characterized by fragile capillaries, poor wound healing, and bone deformity in children.
Vitamin D: A steroid vitamin which promotes absorption and metabolism of calcium and phosphorus. Under normal conditions of sunlight exposure, no dietary supplementation is necessary because sunlight promotes adequate vitamin D synthesis in the skin. Deficiency can lead to osteomalacia in adults and bone deformity (rickets) in children.
Vitamin E: Deficiency can lead to anemia.
Vitamin K: An essential factor in the formation of blood clotting factors. Deficiency can lead to abnormal bleeding.
Nutritions:
For the treatment of nutritional disease, any of the nutrient-related diseases and conditions that cause illness in humans. They may include deficiencies or excesses in the diet, obesity and eating disorders, and chronic diseases such as cardiovascular disease, hypertension, cancer, and diabetes mellitus. Nutritional diseases also include developmental abnormalities that can be prevented by diet, hereditary metabolic disorders that respond to dietary treatment, the interaction of foods and nutrients with drugs, food allergies and intolerances, and potential hazards in the food supply. All of these categories are described in this article. For a discussion of essential nutrients, dietary recommendations, and human nutritional needs and concerns throughout the life cycle, see nutrition, human.
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